Research Paper On Psychological Disorders

Research Paper On Psychological Disorders-82
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Further, mental imagery processes may also underlie the effectiveness of clinical interventions such as imagery re-scripting, imaginal exposure in Cognitive Behavioural Therapy, schema-focused therapy, and cognitive bias modification training.Twin studies report the concordance rate of schizophrenia (the probability that one twin will have the disorder if the other twin does) to be 45 to 60 % for monozygotic (identical) twins, compared to only 10 to 15 % for dizygotic (fraternal) twins. In a comprehensive review of schizophrenia literature, Husted et al. Early environmental exposures influence schizophrenia expression even in the presence of strong genetic predisposition. However, because of the retrospective nature of the study, researchers could not determine which of the many variables associated with famine (e.g. nutrient deficiencies, generalized stress, infection, ingestion of toxic substances, or a combination of those factors) contributed to the increased risk of psychosis.(6) Determining the relative contribution of environmental factors to the onset of schizophrenia is further complicated by the interactions between genetic and environmental variables. (2005) found that different versions of the catecholamine-O-methyl transferase (COMT) gene, which is involved in the regulation of dopamine release in the prefrontal cortex, affected the degree to which adolescent cannabis use was a risk factor for schizophrenia.(7) The genetic contribution to depressive disorders is estimated to be approximately 30 to 40 %.(8) While a variety of environmental characteristics have been identified as risk factors for depression, early life stressors, such as childhood physical or sexual abuse, parental neglect, and loss of a parent, have been shown to significantly increase the probability of developing depression later in life.(9) The effects of early life stressors are influenced by a variety of genes, and the 5-HTTLPR gene, involved in serotonin transporter functioning, has received significant attention. (2003) found that the short version of the gene, which is associated with a reduction in serotonin transporter function, increased the risk of developing depressive symptoms and suicidality following exposure to stressful life events and maltreatment during childhood.(10) However, results from studies of the 5-HTTLPR gene have not been consistent, and Heim et al. Moderation of the effect of adolescent-onset cannabis use on adult psychosis by a functional polymorphism in the catechol-O-methyltransferase gene: longitudinal evidence of a gene-environment interaction. W., Harrington, H., Mc Clay, J., Mill, J., Martin, J., Braithwaite, A., Poulton, R. Influence of life stress on depression: moderation by a polymorphism in the 5-HTT gene.We use cookies to offer you a better experience, personalize content, tailor advertising, provide social media features, and better understand the use of our services.To learn more or modify/prevent the use of cookies, see our Cookie Policy and Privacy Policy.Diagnosing mental-health issues may seem straightforward: Patients discuss their symptoms and a clinician matches those symptoms to a disorder and devises an appropriate treatment.In reality, this view belies the complexity inherent in understanding, classifying, and diagnosing psychiatric phenomena.


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